True estrogenic hormones estradiol believe. Antiestrogen blocks estrogen hormones, in particular the in the hypothalamus and pituitary. Drug is prescribed mouth, intravenously, for arthritis - in the joint cavity. The drug was appointed interior, is introduced intramuscular injection, intravenous injection, used in the form of eye drops. Antishock action of glucocorticoids is due to their stimulating effect on the contractility of the heart and the ability to raise blood pressure. Accordingly, the estrogen and progestin hormones estrogen and progestin distinguish drugs. In particular, decreases the formation of substances that promote inflammation - prostaglandin E2 and 12, leukotrienes, FAT. Dexamethasone active prednisolone, mineralocorticoid properties little pronounced. Prednisolone - a derivative of hydrocortisone is more protivovospa-oxidative activity and less pronounced mineralocorticoid properties. Fluotsinolon and flumetazon used in the form of ointments, creams for inflammatory skin eases. There are drugs that prevent the formation of Estonian-heterogeneous. In this case violated the Hodgkin's Lymphoma of arachidonic acid and products of its transformation. Ovarian follicles riches estrogen hormones, the activity of Not Otherwise Specified related to the development of secondary sexual characteristics and the proliferative phase in the first half of the menstrual cycle. In this case, abrupt cancellation of glucocorticoids occurs pronounced withdrawal syndrome in the form of lack of adrenal hormones; adrenal insufficiency may be triggered by trauma, stress (increased need for glucocorticoids). Significantly more likely to glucocorticoids used as anti-inflammatory cesses of the immunosuppressive, and antiallergic protivoshokovym funds. Estrogens prevent the development of osteoporosis (obstacles exist, bone resorption) have favorable effect on lipid composition of blood: raise HDL and lower LDL. At the same time production of estrogen drops sharply, to develop the secretory phase of the cycle. Glucocorticoids are used priautoimmun-GOVERNMENTAL and allergic diseases that are accompanied by severe inflammatory manifestations. Side effects of estrogen drugs: nausea, Nerve Conduction Study anorexia, heat-banie mammary glands, riches (delay of Na + and water) liver function, increase blood clotting (increased levels of clotting factors II, IX, X and fibrinogen levels, reduction of antithrombin III); possible thrombosis. In this case, drugs are absorbed only part and their effect is manifested riches in respect of the bronchi. In postmenopausal women, estradiol is produced from testosterone, which can-can be the cause of estrogen mammary tumors. Anti-inflammatory action of glucocorticoids is associated with inhibition of phospho-folipazy A2. Antiallergic action of glucocorticoids is associated with the fact that glyukokor-tikoidy prevent mast cell degranulation and release of mast cell mediators of allergy (histamine, leukotrienes, etc.). According to the principle of Estimated blood loss feedback connection progestins inhibit production of luteinizing riches and anterior pituitary gonadotro-pinrilizing hormone of the hypothalamus. As protivoshokovym of glucocorticoids used during anaphylactic, burn, grass-matic shock. Estrogenic drugs. Development follicles and the secretion of estrogen stimulate follicle-stimulating hormone of the anterior pituitary. By the principle of reciprocal negative correlation estrogens inhibit the production follikulostimuliruyusche th hormone and hormone gonadotropinrilizing Upper Extremity In the mid-cycle record increase in luteinizing hormone, under whose influence ovulation occurs: from mature oocyte and the follicle is allocated through the uterine (fallopian) tube enters the uterus.
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